We determined the function of endogenous hydrogen sulfide (H?S) in cerebral vasodilation/hyperemia and early BBB disruption following ischemic heart stroke. CSE knockout (CSE-/-) decreased postischemic cerebral vasodilation/hyperemia but just inhibited Na-F extravasation. An upregulated CBS was within cerebral cortex of CSE-/- mice. Localized treatment with CHH didnt further alter postischemic cerebral vasodilation/hyperemia, but avoided EB… Continue reading We determined the function of endogenous hydrogen sulfide (H?S) in cerebral